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N-acetyl-l-cysteine controls osteoclastogenesis through regulating Th17 differentiation and RANKL in rheumatoid arthritis
Hae-Rim Kim, Kyoung-Woon Kim, Bo-Mi Kim, Kyung-Ann Lee, Sang-Heon Lee
Korean J Intern Med. 2019;34(1):210-219.   Published online 2017 March 13    DOI:

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N-acetyl-l-cysteine controls osteoclastogenesis through regulating Th17 differentiation and RANKL in rheumatoid arthritis
The Korean Journal of Internal Medicine. 2019;34(1):210-219   Crossref logo
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Reciprocal Activation of CD4+ T Cells and Synovial Fibroblasts by Stromal Cell-Derived Factor 1 Promotes RANKL Expression and Osteoclastogenesis in Rheumatoid Arthritis
Arthritis & Rheumatology. 2014;66(3):538-548   Crossref logo
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Interleukin-22 promotes osteoclastogenesis in rheumatoid arthritis through induction of RANKL in human synovial fibroblasts
Arthritis & Rheumatism. 2012;64(4):1015-1023   Crossref logo

Th17 Cells and Rheumatoid Arthritis—From the Standpoint of Osteoclast Differentiation—
Allergology International. 2008;57(2):109-114   Crossref logo
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Plumbagin ameliorates collagen-induced arthritis by regulating Treg/Th17 cell imbalances and suppressing osteoclastogenesis
Osteoarthritis and Cartilage. 2017;25:S417   Crossref logo
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Role of osteopontin in synovial Th17 differentiation in rheumatoid arthritis
Arthritis & Rheumatism. 2010;62(10):2900-2908   Crossref logo
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2-Methoxystypandrone represses RANKL-mediated osteoclastogenesis by down-regulating formation of TRAF6-TAK1 signalling complexes
British Journal of Pharmacology. 2010;161(2):321-335   Crossref logo

Peripheral blood T lymphocytes from patients with early rheumatoid arthritis express RANKL and interleukin-15 on the cell surface and promote osteoclastogenesis in autologous monocytes
Arthritis & Rheumatism. 2006;54(4):1151-1164   Crossref logo
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TLR-3 enhances osteoclastogenesis through upregulation of RANKL expression from fibroblast-like synoviocytes in patients with rheumatoid arthritis
Immunology Letters. 2009;124(1):9-17   Crossref logo
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HMGB1 Promotes the Differentiation of Th17 via Up-Regulating TLR2 and IL-23 of CD14+Monocytes from Patients with Rheumatoid Arthritis
Scandinavian Journal of Immunology. 2012;76(5):483-490   Crossref logo

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