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Clinical characteristics and treatment responses of patients who developed tuberculosis following use of a tumor necrosis factor-α inhibitor
Keun Bum Chung, Eun Young Lee, Jong Pil Im, Sung Koo Han, Jae-Joon Yim
Korean J Intern Med. 2013;28(2):174-179.   Published online 2013 February 27    DOI: https://doi.org/10.3904/kjim.2013.28.2.174

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Clinical characteristics and treatment responses of patients who developed tuberculosis following use of a tumor necrosis factor-α inhibitor
The Korean Journal of Internal Medicine. 2013;28(2):174   Crossref logo
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Sa1119 Clinical Features of Active Tuberculosis Developed During Anti-Tumor Necrosis Factor-α Therapy in Patients With Inflammatory Bowel Diseases
Gastroenterology. 2015;148(4):S-231   Crossref logo
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INCIDENCE OF ACTIVE TUBERCULOSIS WITHIN 1 YEAR AFTER TUMOR NECROSIS FACTOR INHIBITOR TREATMENT ACCORDING TO LTBI STATUS IN PATIENTS WITH INFLAMMATORY BOWEL DISEASE
Respirology. 2018;23:319-319   Crossref logo
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Tumor necrosis factor-alpha inhibitor treatment for sarcoidosis
Therapeutics and Clinical Risk Management. 2008;Volume 4:1305-1313   Crossref logo
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Clinical and High-resolution Computed Tomographic Findings in Five Patients with Pulmonary Tuberculosis Who Developed Respiratory Failure Following Chemotherapy
Clinical Radiology. 2001;56(7):550-555   Crossref logo
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Patients treated with a tumor necrosis factor-α inhibitor are more likely to develop extrapulmonary tuberculosis
The Korean Journal of Internal Medicine. 2013;28(2):159   Crossref logo
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Mycobacterium abscessus infection after use of tumor necrosis factor α inhibitor therapy: case report and review of infectious complications associated with tumor necrosis factor α inhibitor use
Diagnostic Microbiology and Infectious Disease. 2005;53(3):233-238   Crossref logo
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Secondary syphilis following tumor necrosis factor-α inhibitor treatment for rheumatoid arthritis
The Journal of Dermatology. 2011;39(2):199-201   Crossref logo
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Association between the level of circulating bioactive tumor necrosis factor α and the tumor necrosis factor α gene polymorphism at −308 in patients with rheumatoid arthritis treated with a tumor necrosis factor α inhibitor
Arthritis & Rheumatism. 2008;58(5):1258-1263   Crossref logo
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Treatment Persistence and Clinical Outcomes of Tumor Necrosis Factor Inhibitor Cycling or Switching to a New Mechanism of Action Therapy: Real-world Observational Study of Rheumatoid Arthritis Patients in the United States with Prior Tumor Necrosis Factor Inhibitor Therapy
Advances in Therapy. 2017;34(8):1936-1952   Crossref logo
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